Giampaoli S, Palmieri L, Mattiello A, et al. Definition of high risk individuals to optimise strategies for primary prevention of cardiovascular diseases. Nutr Metab Cardiovasc Dis 2005; 15: 79-85.
Greenland P, Knoll MD, Stamler J, et al. Major risk factors as antecedents of fatal and nonfatal coronary heart disease events. JAMA 2003; 290: 891-7.
Khot UN, Khot MB, Bajzer CT, et al. Prevalence of conventional risk factors in patients with coronary heart disease. JAMA 2003; 290: 898-904.
Kannel WB, Dawber TR, Friedman GD, et al. Risk Factors in Coronary Heart Disease. An Evaluation of Several Serum Lipids as Predictors of Coronary Heart Disease; the Framingham Study. Ann Intern Med 1964; 61: 888-99.
Wilson PW. Established risk factors and coronary artery disease: the Framingham Study. Am J Hypertens 1994; 7: 7S-12S.
Wilson PW, D'Agostino RB, Levy D, et al. Prediction of coronary heart disease using risk factor categories. Circulation 1998; 97: 1837-47.
Assmann G, Cullen P and Schulte H. Simple scoring scheme for calculating the risk of acute coronary events based on the 10-year follow-up of the prospective cardiovascular Munster (PROCAM) study. Circulation 2002; 105: 310-5.
Cullen P, Schulte H and Assmann G. Smoking, lipoproteins and coronary heart disease risk. Data from the Munster Heart Study (PROCAM). Eur Heart J 1998; 19: 1632-41.
Seidell JC, Bjorntorp P, Sjostrom L, et al. Regional distribution of muscle and fat mass in men--new insight into the risk of abdominal obesity using computed tomography. Int J Obes 1989; 13: 289-303.
Pollock ML, Hickman T, Kendrick Z, et al. Prediction of body density in young and middle-aged men. J Appl Physiol 1976; 40: 300-4.
Executive Summary of The Third Report of The National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, And Treatment of High Blood Cholesterol In Adults (Adult Treatment Panel III). JAMA 2001; 285: 2486-97.
Assmann G, Carmena R and Cullen P. for the International Task Force for Prevention of Coronary Heart Disease. Coronary heart disease:reducing the risk: the scientific background for the primary and secondary prevention of coronary heart disease: a worldwide view. Nutr Metab Cardiovasc Dis 1998; 205-71.
van der Kooy K, Leenen R, Seidell JC, et al. Waist-hip ratio is a poor predictor of changes in visceral fat. Am J Clin Nutr 1993; 57: 327-33.
International Task Force for Prevention of Coronary Heart Disease, http://www.chd-taskforce.com/slidekit/kit12/slide2_d.htm, last accessed in August 2007.
Grundy SM, Pasternak R, Greenland P, et al. Assessment of cardiovascular risk by use of multiple-risk-factor assessment equations: a statement for healthcare professionals from the American Heart Association and the American College of Cardiology. Circulation 1999; 100: 1481-92.
Janssen I, Katzmarzyk PT, Ross R, et al. Fitness alters the associations of BMI and waist circumference with total and abdominal fat. Obes Res 2004; 12: 525-37.
Assmann G, Cullen P and Schulte H. The Munster Heart Study (PROCAM). Results of follow-up at 8 years. Eur Heart J 1998; 19 Suppl A: A2-11.
Assmann G, Schulte H, von Eckardstein A, et al. High-density lipoprotein cholesterol as a predictor of coronary heart disease risk. The PROCAM experience and pathophysiological implications for reverse cholesterol transport. Atherosclerosis 1996; 124 Suppl: S11-20.
Assmann G. Calculating global risk: the key to intervention. Eur Heart J Suppl F 2005; F9-F14.
Wang J, Thornton JC, Kolesnik S, et al. Anthropometry in body composition. An overview. Ann N Y Acad Sci 2000; 904: 317-26.
Cooper JA, Miller GJ and Humphries SE. A comparison of the PROCAM and Framingham point-scoring systems for estimation of individual risk of coronary heart disease in the Second Northwick Park Heart Study. Atherosclerosis 2005; 181: 93-100.
Empana JP, Ducimetiere P, Arveiler D, et al. Are the Framingham and PROCAM coronary heart disease risk functions applicable to different European populations? The PRIME Study. Eur Heart J 2003; 24: 1903-11.
Evaluation of the association between the first observation and the longitudinal change in C-reactive protein, and all-cause mortality. Heart 2008;94:457-62